A Review of the Theoretical Unified Approach to Podiatric Biomechanics in Relation to Foot Orthoses Therapy

Similar documents
The Unified Theory of Foot Function

Recent Advances in Orthotic Therapy for. Plantar Fasciitis. An Evidence Based Approach. Lawrence Z. Huppin, D.P.M.

Giovanni Alfonso Borelli Father of Biomechanics

New research that enhances our knowledge of foot mechanics as well as the effect of

Purpose A patented technology designed to improve stability of the human foot.

Treating Foot Pain in Alpine Skiers with

SECTION 4 - POSITIVE CASTING

ATHLETES AND ORTHOTICS. January 29, 2014

PLANTAR FASCIITIS. Points of Confusion. TREATING SUBCALCANEAL PAIN: Who gets the best outcomes?

Introduction to Biomechanical Evaluation Qualitative Biomechanics Peter G. Guy B.Sc., D.Ch.

Throw the Baby Out with the Bath Water? Challenging the Paradigm of Root Biomechanics

Case Study: Chronic Plantar Heel Pain/Plantar Fasciopathy. CASE STUDY PRESENTATION by Resonance Podiatry and Gait Labs

10 Orthotic Modifications You Can Perform in the Office. These simple fixes will lead to happier patients. BY DIANNE MITCHELL, DPM

Foot mechanics & implications on training, posture and movement

Clinical Observational Gait Analysis

FOOT FUNCTIONING PARADIGMS

Foot Biomechanics Getting Back to the Base

Afoot. What s VASYLI. Biomechanical foot function: a Podiatric perspective. Introduction: Anatomical structure and function. Continued on Page 2

a podiatric perspective: part 2

Normal Gait and Dynamic Function purpose of the foot in ambulation. Normal Gait and Dynamic Function purpose of the foot in ambulation

The Problem. An Innovative Approach to the Injured Runner. Dosage. Mechanics. Structure! Postural Observations. Lower Quarter Assessment

Blomechamcal foot funcuon: a podiatric perspective: part 1

Customized rocker sole constructions

PLANTAR FASCIITIS why does sleep hurt my feet?

Ankle biomechanics demonstrates excessive and prolonged time to peak rearfoot eversion (see Foot Complex graph). We would not necessarily expect

Custom Ankle Foot Orthoses

Longitudinal Arch Angle (LAA): Inter-rater reliability comparing Relaxed Calcaneal Stance with Toe Off

10/24/2016. The Puzzle of Pain NMT and the Dynamic Foot Judith DeLany, LMT. Judith DeLany, LMT. NMTCenter.com. NMTCenter.com

Ice skating in all its various forms. Living on the Edge: The Biomechanics of Power Skating

A Comparison of Different Casting Methods and How They Affect the Orthotic Device Produced.

Foot Posture Biomechanics and MASS Theory

Chapter 1 - Injury overview Chapter 2 - Fit for Running Assessment Chapter 3 - Soft Tissue Mobilization... 21

'Supporting' the Foot 2 May 2002

Assessment of the Foot in Relation to Gait Dysfunction and Injury Day 1 Paul Harradine MSc FFPM FRPS (glasg) FCPM CertEd

Evidence-Based Medicine: Foot Imaging for Custom Functional Foot Orthoses

Normal Biomechanics of the Foot and Ankle

The Starting Point. Prosthetic Alignment in the Transtibial Amputee. Outline. COM Motion in the Coronal Plane

PHASIC POSTING Putting The Function Into Foot Orthoses. Bob Longworth MSc DPodM Lee Short MSc

Purpose. Outline. Angle definition. Objectives:

Diabetes and Orthoses. Rob Bradbury Talar Made

Normal and Abnormal Gait

Effect of Ankle Dorsiflexion Range of Motion on Rearfoot Motion During Walking

VS. THE OLD ROOT THEORY

Dynamix Ankle Foot Orthoses Range

The Effects of Vacuum-Molded Orthotics on Lower Extremity Overuse Injuries

Pathomechanics of Structural Foot Deformities

PATHOLOGICAL CONDITIONS REQUIRING THE USE OF CUSTOMIZED LASTS

Continuing Medical Education Continuing. Medical Education

Foot orthoses and lower extremity pathology

As a physiotherapist I see many runners in my practice,

video Outline Pre-requisites of Typical Gait Case Studies Case 1 L5 Myelomeningocele Case 1 L5 Myelomeningocele

ASSESMENT Introduction REPORTS Running Reports Walking Reports Written Report

The Peacock Press Test

Sample Biomechanical Report

Assessments SIMPLY GAIT. Posture and Gait. Observing Posture and Gait. Postural Assessment. Postural Assessment 6/28/2016

Shin Splints and Forefoot Contact Running: A Case Report

Aeris Performance 2. Product Manual

Biomechanical Foot Orthotics: A Retrospective Study

Aeris Activity. Product Manual

AllPro Foot. Posterior Mount Product Manual

Comparison of Langer Biomechanic s DynaFlange to Traditional Legacy Rearfoot Posted Orthotics

Case Report: The Infant Flatfoot

A bit of background. Session Schedule 3:00-3:10: Introduction & session overview. Overarching research theme: CPTA

Product catalog Top models

The Human Foot. The Three Major Sections of the Foot

COMPENSATORY EFFECTS OF FOOT DEFORMITY:

Running injuries - what are the most important factors

Smita Rao PT PhD. Judith F. Baumhauer MD Josh Tome MS Deborah A. Nawoczenski PT PhD

EDUCATION COURSES. Stride. Initial Swing (high knee) Mid stance Toe off Mid swing Initial contact

1. Hip flexion Muscles: Iliopsoas (psoas major + iliacus)

The scientific basis for the use of biomechanical foot

A Functional Approach to Improving Ankle Dorsiflexion. Knock on Effects

The Lateralized Foot & Ankle Pattern and the Pronated Left Chest

video Purpose Pathological Gait Objectives: Primary, Secondary and Compensatory Gait Deviations in CP AACPDM IC #3 1

Element DS. Product Manual

Gait analysis through sound

Plantar fasciitis: identify & overcome

BIOMECHANICAL ASSESSMENT & ORTHOTIC SALES SCRIPTS

The relationship between the structure of the foot and its

Running from injury 2

THE ANKLE-HIP TRANSVERSE PLANE COUPLING DURING THE STANCE PHASE OF NORMAL WALKING

SAPPHIRE PHYSICAL THERAPY

Rob Burke B.Sc. KIN and Reggie Reyes B.Sc. KIN August 2002

Athlete Profiling. Injury Prevention

Running Injuries in Adolescents Jeffrey Shilt, M.D. Part 1 Page 1

12/4/2010 3:10 / 3:40

10/22/15. Walking vs Running. Normal Running Mechanics. Treadmill vs. Overground Are they the same? Importance of Gait Analysis.

RUNNING SHOE STIFFNESS: THE EFFECT ON WALKING GAIT

Notes Session #2. The second gravity organization system is the relationship of the feet with the ground.

Is it important to position foot in subtalar joint neutral position during non-weight-bearing molding for foot orthoses?

Back Pain in swimmers Aetiology

The DAFO Guide to Brace Selection

AllPro Foot. Product Manual

CHAPTER IV FINITE ELEMENT ANALYSIS OF THE KNEE JOINT WITHOUT A MEDICAL IMPLANT

line insoles Introducing our new X-Line insole range INSOLES Condition Specific

Challenging the foundations of the clinical model of foot function: further evidence that

THE INFLUENCE OF SLOW RECOVERY INSOLE ON PLANTAR PRESSURE AND CONTACT AREA DURING WALKING

Assessment of Subtalar Joint Neutral Position: Study of Image Processing for Rear Foot Image

THE EFFECTS OF ORTHOTICS ON LOWER EXTREMITY VARIABILITY DURING RUNNING. Samuel Brethauer

Steffen Willwacher, Katina Fischer, Gert Peter Brüggemann Institute of Biomechanics and Orthopaedics, German Sport University, Cologne, Germany

Transcription:

ORIGINAL ARTICLES A Review of the Theoretical Unified Approach to Podiatric Biomechanics in Relation to Foot Orthoses Therapy Paul Harradine, MSc* Lawrence Bevan, BSc Background: Diverse theories of orthoses application have evolved with the continual development of podiatric biomechanics and orthotic management. This theoretical disparity can lead to confusion in clinical, educational, and research situations. However, although approaches are varied, the common consensus is that foot orthoses outcomes are generally positive. Methods: Three main podiatric theories exist: the foot morphology theory, the sagittal plane facilitation theory, and tissue stress theory. By researching the available literature, the perspectives of all three theories are summarized, emphasizing areas of conflict and agreement. Results: Through a unified theory, we introduce a premise by which the similar orthotic outcomes obtained from the three main podiatric theories may be explained. Conclusions: It remains up to the individual podiatric physician to decide which method to use to prescribe a foot orthosis. It may be of benefit to encompass all approaches rather than be dogmatic or exclusive. (J Am Podiatr Med Assoc 99(4): 317-325, 2009) Podiatric physicians have been using in-shoe appliances and varied theories and therapies to treat gaitrelated symptoms since the profession began to develop in the 18th century. 1 More recently, three main theories have become established in the podiatric literature in relation to treating foot-related lower-quadrant symptoms. The subtalar joint neutral (STJN) theory, tissue stress (TS) theory, and sagittal plane facilitation (SPF) theory make up the most accepted approaches to the foot in relation to gait dysfunction. 2, 3 Although these theories appear to be diverse in their application, the common consensus of outcomes for treatment of lower-limb symptoms from any of these paradigms is generally positive. 4-18 We present theoretical standpoints and methods of orthotic prescription; critical review of these theories is available elsewhere. 2, 3 *The Podiatry and Chiropody Centre, Portsmouth, Hants, United Kingdom. Gloucestershire Podiatry Department, St. Paul s Medical Centre, Cheltenham, United Kingdom. Corresponding author: Paul Harradine, MSc, The Podiatry and Chiropody Centre, 77 Chatsworth Ave, Portsmouth, Hants, PO6 2UH United Kingdom. (E-mail: podiathing@yahoo.co.uk) Foot Morphology Theory Between 1958 and 1959, Merton L. Root, DPM, 19 pioneered functional foot orthoses, conducted hundreds of biomechanical assessments, and began to define the STJN position. The theory that he and his colleagues created is based on the premise that a foot is functioning normally when STJN position occurs immediately after heel strike and at the end of the midstance phase of gait. 20 Foot morphology (FM) was characterized and referenced to this STJN position, 21 and the relationship between this and normal or abnormal foot function was established. 20 Although Dr. Root may have been developing orthoses as part of his clinical practice, 19 no further descriptive text on foot orthotic prescription or manufacture was ever made available. However, many authors have cited Dr. Root in their interpretation of their own texts and literature on foot orthoses, often using terminology such as Rootian or Modified Rootian foot orthoses. 22-30 It may be unwise to assume that Dr. Root would agree with the interpretation of his work, although all of these contributors base their description of or- Journal of the American Podiatric Medical Association Vol 99 No 4 July/August 2009 317

thotic therapy on FM and the STJN position in some element. The premise of this model of management seeks to identify FM that is abnormal, eg, forefoot varus, and prescribe an orthotic device to prevent subsequent abnormal joint compensatory motion eg, excessive subtalar joint (STJ) pronation. 19, 20, 24-26, 30 It may be fair to assume that this method of podiatric assessment and treatment is the most popular biomechanical approach to foot function and orthoses used worldwide by both podiatrists and other professions. 31-33 The majority of texts that describe orthoses in relation to Roots work agree with the following prescriptive methodology, which is generally taken from the most comprehensive available literature on FM theory orthoses. 22 The FM theory orthosis is designed to balance a foot deformity with posting applied to a rigid bespoke shell. Prescription protocol begins with a cast of the foot in a nonweightbearing neutral position. 34 The shape of the neutral cast is of prime importance, because it is essential to capture the correct forefoot-to-rearfoot alignment and calcaneal inclination angle. The cast is then angled with an intrinsic forefoot post to place the bisection of the heel at the required position. The degree of posting to achieve this required position is calculated by taking the value of a patient s neutral calcaneal stance position (NCSP) and subtracting a set number of degrees in order to allow normal pronation. The height of the STJ axis is used to determine the amount of pronation to be allowed. The rearfoot post is ground so that it is angled an additional 4 varus for an average STJ axis. A high STJ axis rearfoot post allows 2 of motion, and the low STJ axis post allows 6, 35 which will control the movement of the foot from NCSP to the prescribed pronated position. To maintain the posting and shape of the orthosis, only rigid materials are recommended, such as acrylic or carbon fiber plus an acrylic rearfoot post. The shell is classically cut to 25% of the width of the first ray. Sagittal Plane Facilitation Theory Howard Dananberg, DPM, first published his theories of SPF in 1986. 36 He and his colleagues have since developed a theory that highlights the importance of the foot as a pivot that rocks forward from heel to toe, thereby allowing adequate hip extension leading up to the propulsive phase of gait. He proposes that this hip extension allows a normal stride and therefore an efficient and erect gait. 5, 36-39 Functional hallux limitus and ankle equinus are two examples of pathology that can restrict foot movement and result in what Dananberg 40 terms a sagittal plane blockade. Ankle equinus is stated to be the inability to dorsiflex to 100, and functional hallux limitus is defined as a first metatarsophalangeal joint (MTPJ) that structurally has a normal range of motion but is unable to dorsiflex adequately at the appropriate time in gait. Dananberg has mostly related SPF theory to more proximal posture-related problems, such as low-back pain, 5, 39, 40 although it is possible to use this theory to explain foot pains and abnormalities. Sagittal plane facilitation theory is now well published, and like the FM theory, is apparently being well accepted by other professions. 39 Orthotic prescription is based upon the premise of informed trial and error using video gait analysis and in-shoe pressure system measures. Therefore, the means of determining what posting, shell thickness, heel raise, and so forth is determined without reference to the forefoot-to-rearfoot relationship or axis height as in the FM model. Dananberg 5, 41 also cites the use of shell modifications, such as cut-outs beneath the first ray and specific forefoot extensions to encourage medial propulsion. In fact, the amount of posting is relatively very small, eg, 1, 41 and potentially totally different from what may seem necessary from a standard static examination. Although a specific SPF theory outcome study is available, 5 the actual prescription method remains sparsely documented. Dananberg has not yet produced a step-by-step guideline of his methodology, making it difficult for practitioners to replicate this often technologically intricate approach. Experience guides the choice of shell modifications. 41 Dananberg has always stated orthoses should be fabricated on functional rather than static examination. However, there is one reference available in which there appears to be an attempt to link SPF theory prescription methodology with deformities identified by Root et al. 42 Sample prescriptions are provided based on three vaguely defined foot types. This appears to be the closest we get to a simple prescription guideline. Table 1 shows a summary of treatment options from the SPF theory perspective compiled from Dananberg s various publications and lectures. 5, 41-43 Tissue Stress Theory Kevin Kirby, DPM, 44 first published work on variations in the STJ axis in 1987. This model is based on assessment of the moments across the STJ and methods of changing these to decrease stress upon anatomical structures. 44-46 Injured structures are identified and pathology is mechanically related to foot function. Fuller 47 recently applied the concept of the windlass mechanism and center of pressure 48 to Kirby s earlier work. This approach employs the application 318 July/August 2009 Vol 99 No 4 Journal of the American Podiatric Medical Association

Table 1. Summary of Treatment Options from the SPF Perspective5, 41-43 Orthoses are custom made from foot impressions, but the method of casting is not highlighted. Dananberg has recently been part of a venture to produce a noncustom prefabricated appliance, the Vasyli Howard Dananberg (VHD) orthotic (Vasyli Medical, Queensland, Australia), which may eliminate the need for casting. Use static and dynamic assessment to establish any need for correcting a leg length difference with heel raises. Manipulate areas of reduced motion such as the first metatarsophalangeal joint and talocrural joint, if required. Supply firm heel raises to reduce the effects of an ankle equinus. Use first-ray shell excisions if there is functional hallux limitus. The size of the cut-out is dependent on dynamic findings. Use a kinetic wedge and digital platform if necessary. Dananberg believes a rearfoot post can be potentially harmful due to encouraging lateral avoidance and advocates the use of flat rearfoot posts. Rearfoot posting is recommended in situations such as a medial heel strike; the maximum required is considered to be 3. Identify and strengthen weakness in muscles that are pivotal in the SPF theory such as the peroneus longus or tibialis posterior. Depending on dynamic findings, post the forefoot no more than 3 varus or valgus. Use semirigid-to-flexible materials. Both allowing and controlling motion is a central concept. If early heel lift is a problem, use soft poron heel lifts as dampeners under the rearfoot post. Abbreviation: SPF, sagittal plane facilitation. of physical laws such as moments, levers, stress, and strain curves. Tissue stress theory is based more on the concept of kinetics as opposed to kinematics of gait. The central concept is that pronation or supination does not cause harm but stopping the pronation or supination does. If the center of pressure is medial to the STJ axis during gait, a supinatory moment will be applied to the STJ. The opposite will also occur. If the center of pressure is lateral to the STJ axis, a pronatory moment will be applied to the STJ. For rotational equilibrium to occur, structures opposing these moments must apply a moment of the same magnitude. For example, the plantar fascia and the tibialis posterior would oppose a pronatory moment across the STJ axis. A medial or lateral shift in the STJ axis will result in a disturbance of this equilibrium, and undesirable motion will occur unless a structure can increase the opposing moment. The strain that this imposes on opposing structures, such as the tibialis posterior, may exceed its loading capacity and result in injury. 44-48 Symptom reduction appears to dictate treatment outcome rather than the success or otherwise of placing the foot in an ideal position. 49, 50 Foot-related musculoskeletal injury is treated with orthoses to reduce the abnormal forces on injured structures by applying appropriate moments to the STJ. 48, 50 Kirby 46 has contested the criteria for normality set out by Root et al, 20, 21 proposing it to be irrelevant to achieving normal foot function with an orthosis. Kirby furthers this by commenting that a foot resting very near to STJN in relaxed bipedal stance actually exhibits excessive supination moments in gait. A moderately pronated foot in stance is considered more of a normal position. 46 Orthotic prescription choices are forefoot posting, rearfoot posting, and forefoot extensions in a valgus or varus orientation. Negative casts are often modified at the time of impression, depending on the required shell shape, to apply the correct moments to the foot, eg, the first ray is dorsiflexed or plantarflexed to alter forefoot to rearfoot alignment. 49 Large degrees of forefoot posting can be used, up to 5 to 10. 46, 50 The magnitude of pathologic moments dictates the amount of posting required rather than modifying a cast or orthotic shell based on the STJN position. The general width of the orthoses is not specified, although they appear commonly wider than FM theory prescriptions. 50 In contrast to the FM theory, a change in magnitude of forces, not in joint position, is required to reduce symptoms. Greater orthotic reaction force is required if the ground reaction force is producing abnormal moments. To supply this increased orthotic reaction force, Kirby designed and published a shell modification called the heel skive. 51 By modifying the positive cast, the heel skive can be applied, as required, to the medial or lateral aspect of the STJ axis. As with the SPF theory, there is no actual prescription protocol available for the production of orthoses based on the TS theory, although more literature is available. 50, 52 A Unified Theory It is reasonable to assume that no clinician would continue to use a theory that was not working to relieve their patients symptoms. Rationally there must be beneficial aspects of treatment from FM, SPF, and TS theory perspectives. Although the three main podiatric biomechanics paradigms conflict in principle (Table 2), all demonstrate an agreement that the foot essentially has three main areas of function 20, 40, 46, 50 : 1) to be stable and maintain a congruent structure Journal of the American Podiatric Medical Association Vol 99 No 4 July/August 2009 319

Table 2. Examples of the Underpinning Theoretical Differences Between Podiatric Foot Function Paradigms Theoretical Perspective Foot Morphology Theory Sagittal Plane Facilitation Theory Tissue Stress Theory Criteria for normalcy The STJ passes through neutral at key stages of the gait cycle. The foot functions as a pivot allowing adequate hip extension and correct posture. The foot functions in a way that does not result in abnormal tissue stress and injury. Casting methodology The foot is cast in STJN, unless a large deformity is a contraindication. Casting methods are not documented, although recent noncustom orthoses from this theory may eliminate the need for casting. The positive cast is modified when taken to supply the shell shape required to apply the correct forces to the foot. Orthoses aim To prevent abnormal joint compensation and place the foot into its normal position for key stages of the gait cycle. To allow the foot to work successfully as a pivot and facilitate sagittal plane motion. To reduce abnormal stress upon symptomatic structures. Abbreviations: STJ, subtalar joint; STJN, subtalar joint neutral. through the stance phase; 2) to allow the leg to pivot over the point of ground contact, permitting a normal stride; and 3) to allow internal and then external rotation of the leg in relation to the ground through STJ pronation and supination. A common underlying corrective mechanism may exist, or there may be more than one way to improve symptoms with the use of orthoses. We present a theory to explain normal and abnormal foot function, which can be used to unify and explain benefits reportedly obtained from the three established theories. For the purpose of this article, we discuss methods of normalizing foot function to improve symptoms rather than the limitation of normal motion that can be provided by orthoses to relieve specific symptoms, for example, the Morton s extension. 53 Theoretical Mechanism for Normal Foot Function As the foot hits the ground, initial double-limb stance occurs. At this stage, the contact phase lower limb, in relation to the ground, is internally rotating. 54 To allow this internal rotation, the STJ pronates and the arch lowers. As the arch lowers, it becomes longer and structures that originate proximal and insert distal to the midtarsal joint have increased tension. Relevant examples of these structures are presented in Table 3. This increased tension from these structures applies a longitudinal compressive force through the convex and concave joints of the midtarsus, theoretically close-packing these joints and increasing the congruent stability of the foot. In specific relation to the plantar fascia, a reverse windlass mechanism has been proposed. 55, 56 Not only does the increased ten- sion in the plantar fascia increase the stability of the midtarsus, it also pulls the digits to the ground because of the insertion of the plantar fascia into the digits (Fig. 1). The normal amount of pronation that occurs with internal leg rotation at contact phase therefore supplies stability of the foot that is essential for a normal gait cycle. Through midstance, the leg begins to externally rotate in relation to the ground. 54 This rotation requires STJ supination. With this supination, the arch begins to rise and therefore the origin and insertion of the structures responsible for aiding the congruent stability of the foot become closer and tension could be lost. This occurs with heel lift, a stage in gait in which the body s center of mass progresses anterior to the ankle over the midtarsal joint. 54 This progression on center of mass creates a peak moment that attempts to lower the arch. The ability of the foot to resist these bending moments and maintain arch raising is paramount. Unless the slack in any of the plantar structures can be taken up, stability may be lost. The windlass effect was first described by Hicks 57 in 1954 and has more recently been expanded on from different perspectives by Fuller 47 and Dananberg. 40 Involved anatomy is that of the medial arch and medial band of the plantar fascia. The medial band of the plantar fascia originates from the medial tubercle of the calcaneus and inserts distally into the base of the proximal phalanx and sesamoid bones. 58 During closed kinetic chain in a foot with a normal structure, dorsiflexion of the hallux will tighten the plantar fascia because of the plantar fascia being wound around the first metatarsal head. This is analogous to a cable being wound around a windlass. 56 This effectively draws the head of the first metatarsal 320 July/August 2009 Vol 99 No 4 Journal of the American Podiatric Medical Association

Table 3. Examples of Structures with Theoretically Increased Tension with Lowering of the Arch via STJ Pronation Structure Origin and Insertion Plantar fascia Long calcaneocuboid ligament Short calcaneocuboid ligament Calcaneonavicular ligament Peroneal longus tendon Flexor digitorum longus tendon Flexor hallucis longus tendon Posterior tibial tendon Abbreviation: STJ, subtalar joint. Medial tubercle of the calcaneus inserting into the digits. The medial band is the most substantial and inserts into the proximal phalanx of the hallux. Plantar aspect of the calcaneum to the plantar aspect of the cuboid and the bases of the third, fourth, and fifth metatarsals. Plantar anterior tubercle of the calcaneum to the adjoining aspect of the cuboid. Anterior margin of the sustentaculum tali to the inferior surface and the tuberocity of the navicular. Lateral upper two-thirds of the fibula. The tendon runs forward on the lateral aspect of the calcaneum around the lateral aspect of the cuboid and inserts into the medial cuneiform and base of the first metatarsal. Posterior surface of the tibia to the bases of the distal phalanges of the lesser digits. Posterior surface of the tibia to the base of the distal phalanx of the hallux. Posterior surface of the tibia, fibia, and interosseous membrane to the tuberocity of the navicular and plantar aspects of the cuboid, cuneiforms, and bases of the second, third, and fourth metatarsals. and calcaneus together, causing the foot to shorten and the arch to raise (Fig. 2). During propulsion, most of the weightbearing is borne through the medial column of the foot while the leg is externally rotating and the arch is rising and shortening. For the heel to lift, the hallux therefore should dorsiflex. This winds the windlass and maintains tension in the plantar fascia, creating a compressive force across the foot. The foot therefore uses the internal rotation of the leg to aid in its own stability by increasing plantar structure tension with pronation through contact and early midstance. As the foot pivots over the hallux, the leg externally rotates and midfoot stability is maintained through the windlass mechanism. Although the plantar fascia may be only one of the many structures involved in maintaining foot structure in relaxed bipedal stance, 59 it is theoretically essential in maintaining midtarsus stability during resupination at heel lift. Theoretical Mechanism for Abnormal Foot Function Failure to be Stable and Maintain a Congruent Structure through Stance Phase If moments resisting STJ pronation and arch lowering are not of significant magnitude, abnormal pronation may occur and the midtarus may move to a position of poor congruency through malalignment. 60, 61 Factors Figure 1. Simple model demonstrating the reverse windlass mechanism. Figure 2. Simple model demonstrating the dynamic windlass mechanism. Journal of the American Podiatric Medical Association Vol 99 No 4 July/August 2009 321

that either increase pronatory moments or decrease the foot s ability to resist pronatory moments may be intrinsic to the foot (eg, a medially deviated axis 46 ), extrinsic to the foot (eg, weak lateral hip rotators 62 ), or even transient to the foot (eg, ligamentous laxity in pregnancy 63 ). In addition, for stability to be maintained after heel lift, the windlass mechanism needs to apply tension to the plantar fascia. If this fails, the midtarsus will be unstable at heel lift and unable to resist the bending moment applied as the heel is pulled off the floor. The lowering of the arch at heel lift is analogous to STJ pronation rather than the required supination. For the windlass to function effectively, dorsiflexion of the hallux through medial column propulsion must occur. If a limitation of first MTPJ dorsiflexion is present, whether structural or functional, a lack of windlass mechanism may arise with resultant sequelae. A common compensation mechanism may be to propulse laterally rather than medially, 40, 64 which has been stated not only to fail to wind the windlass but also to be less efficient in propulsion. 65 Although causes of a structural hallux limitus are well discussed in the literature, 66-68 the etiology of functional hallux limitus is less reported. Two possible etiologies are a prolonged reverse windlass and functional bony restriction of the first MTPJ. A prolonged reverse windlass occurs as a result of excessive pronation moments at the STJ. These excessive moments may be attributable to a myriad of causes such as a forefoot varus, tibial varum, or weak lateral hip rotators. The resultant prolonged reverse windlass results in drawn out plantarflexory moments at the first MTPJ when dorsiflexion should be occurring. Such increased plantarflexory moments will therefore impede hallux dorsiflexion and reduce the ability of the foot to propulse through the first MTPJ. Pressure may remain lateral, engaging the foot in inefficient propulsion. 64 This, in turn, inhibits the arch rising because of the lack of a windlass mechanism. In a functional bony restriction of the first MTPJ, dorsiflexion of the first ray impedes the ability of the first MTPJ to extend. 69 Pronation will lead to dorsiflexion of the first ray through increased ground reaction forces to the medial column of the foot. 70 This will limit the ability of the foot to pivot over the first MTPJ, leading to sequelae as described with a prolonged reverse windlass mechanism. The resultant effect is similar to that of a structural hallux limitus. Other possible causes of functional hallux limitus include sesamoid apparatus failure and fixed elevated first ray abnormalities. Failure to Allow the Foot to Pivot and Permit Normal Stride The three rockers (round underside of the heel, ankle dorsiflexion, and hallux dorsiflexion) need to be correctly timed to coincide with proximally occurring motion. For example, as the center of mass advances and the hip extends, the heel must lift appropriately. 54 Failure of the first MTPJ to extend at this vital moment will impede heel lift and limit hip extension with consequences up the kinetic chain. 40 For appropriate timing of the foot rocker motions, it is essential that the foot is stable under load, as discussed previously. Compensation pathways such as a flattened lordosis and lack of hip and knee extension have been stated in the literature. 5, 39, 42 Failure to Allow Internal and External Rotation of the Leg in Relation to the Ground Through STJ Pronation and Supination If the STJ has an inadequate range of pronation to allow normal internal leg rotation at the hip (eg, triple arthrodesis), normal lumbar and pelvic mechanics will not occur. The reverse is also true: if there is a lack of internal rotation at the hip, a normal amount of STJ pronation may not occur to adequately tense the plantar structures of the foot and engage the reverse windlass. An example of this may be a lack of internal hip rotation demonstrated in osteoarthritic hips. 71 From midstance, in relation to the ground, the leg externally rotates and applies a supinatory moment to the STJ. For the leg to externally rotate, the foot must supinate, and the supinatory moment must be greater than the pronatory moment across the STJ. In abnormal situations, this may not occur and can be attributable to a lack of applied supinatory moments or increased pronatory moments. There are several methods by which compensation may occur. The leg may simply remain internally rotated, or if the friction coefficient between the floor and the foot is overcome, the foot may be seen to rapidly abduct and allow external hip rotation without resupination. This has been called an abductory twist. 72 Orthotic Prescription Based on the Unified Theory The fundamental reason for prescribing functional foot orthoses is to improve symptomatic gait dysfunction. This may be caused by one or a combination of factors resulting in a failure of the foot to function as a stable pivot or to allow normal transverse plane hip 322 July/August 2009 Vol 99 No 4 Journal of the American Podiatric Medical Association

motion. It may be argued that symptomatic gait dysfunction was achieved through previous podiatric theories, with combinations of negative cast shapes, varus or valgus posting, or modifications in orthotic width or extension. For example, TS theory orthotic prescription would reduce the prolonged windlass by using varus posting. Sagittal plane facilitation theory would reduce functional bony restriction of the first MTPJ by using cut-outs in the shell beneath the first ray and a kinetic wedge. The FM theory may improve both of the above abnormalities by varus posting and a shell cut narrow to 25% of the width of the first ray. The FM, SPF, and TS theories would all aid the windlass mechanism and improve the foot s ability to function as a stable congruent pivot and assist normal transverse plane hip rotation. It can, therefore, be demonstrated by the simple examples above that all three theories can be conflicting in nature, and yet coadunate in treatment, by using a unifying theory. This can also be linked to specific symptom relief. Plantar fasciitis is one of the most common musculoskeletal injuries presenting to the podiatric physician and is commonly treated with orthoses. 14 For the purpose of discussion, we shall assume that the orthoses work by reducing excessive tension through the plantar fascia, correcting timing of both the reverse and normal windlass mechanisms, thereby allowing normal gait to occur. The orthotic prescription based on the FM theory applies posting for feet that demonstrate abnormal compensation. Beneficial effects may therefore be explained by the reduction of abnormal compensation, allowing the foot to function around the STJN. Alternatively, the device may be seen to reduce strain in the plantar fascia due to reduction of excessive pronatory moments placed across the STJ during the reverse windlass phase. Therefore, orthoses make it easier for the hallux to dorsiflex and normal windlass to occur because of a reduction of plantar fascia stress. In addition, the classic FM theory orthotic appliance is often cut narrow, so that only 25% of the first ray width remains. This decreases dorsiflexory moments on the first ray allowing the hallux to dorsiflex more easily and aid first-ray propulsion. Sagittal plane facilitation theory and TS theory achieve this benefit in similar ways while employing particular methods of orthotic construction or modifications. Sagittal plane facilitation theory uses the kinetic wedge and first-ray cut-outs as well as minor posting. This reduces strain on the plantar fascia, decreases dorsiflexory moments on the first MTPJ via the cut-out, and increases dorsiflexory moments on the first MTPJ via the kinetic wedge. Tissue stress theory advocates the use of greater posting and the medial heel skive. These increase supinatory moments across the STJ axis and unload excessive stress in the plantar fascia. Whether one method is better than the other remains undetermined. Certain orthoses working by a particular orthotic prescription theory may be better at correcting certain gait abnormalities or symptoms than others. However, one might suggest that the relief of symptoms has always occurred through the improvement of normal foot mechanisms, and the method or approach to achieve this is inconsequential. Such conclusions will depend on research outcomes not yet available to us. Conclusions We have attempted to summarize the available literature on the three main podiatric theories of gait dysfunction and have proposed a possible unification theory. Our theory may be seen as an over-simplification of orthotic prescription, but it may explain the fundamental principles behind the apparent universal success of different approaches to orthotic therapy. We have not intended to dismiss or prioritize one method of assessment or treatment over another. Theoretical issues highlighted within podiatric biomechanics are not unique. Other clinical specialties have conflicting theories of musculoskeletal treatment and many of these lack evidence. 73, 74 Until greater research is available, each practitioner should decide which method to use. Podiatric physicians who encompass all approaches may have benefits over those who are more dogmatic. Other determining factors such as bulk of theory-based appliances, material durability, and sporting suitability have not been discussed. As far as the authors are aware, the perfect foot orthosis has not yet been conceived. It is hoped that as evidence-based practice becomes more established, more podiatric physicians and students will undertake areas of required research related to the areas highlighted in this article. Financial Disclosure: None reported. Conflict of Interest: None reported. References 1. LEE WE: Podiatric biomechanics. An historical appraisal and discussion of the Root model as a clinical system of approach in the present context of theoretical uncertainty. Clin Podiatr Med Surg 18: 555, 2001. 2. HARRADINE P, BEVAN L, CARTER N: An overview of podiatric biomechanics theory and its relation to selected gait dysfunction. Physiotherapy 92: 122, 2006. Journal of the American Podiatric Medical Association Vol 99 No 4 July/August 2009 323

3. HARRADINE PD, BEVAN LJ, CARTER N: Gait dysfunction and podiatric therapy: part 1. Foot-based models and orthotic management. Br J Podiatry 6: 5, 2003. 4. HARRADINE PD, JARRET J: Podiatric biomechanics: the efficacy of a service within the NHS. Foot 11: 15, 2001. 5. DANANBERG HJ, GUILIANO M: Chronic low-back pain and its response to custom-made foot orthoses. JAPMA 89: 109, 1999. 6. LANDORF K, KEENAN A: Efficacy of foot orthoses: what does the literature tell us? JAPMA 90: 149, 2000. 7. BLAKE RL, DENTON JA: Functional foot orthoses for athletic injuries: a retrospective study. JAPMA 75: 359, 1985. 8. DONATELLI R, HURLBERT C, CONAWAY D, ET AL: Biomechanical foot orthotics: a retrospective study. J Orthop Sports Phys Ther 10: 205, 1988. 9. GROSS ML, DAVLIN LB, EVANSKI PM: Effectiveness of orthotic shoe inserts in the long-distance runner. Am J Sports Med 19: 409, 1991. 10. THOMPSON JA, JENNINGS MB, HODGE W: Orthotic therapy in the management of osteoarthritis. JAPMA 82: 136, 1992. 11. FERGUSON H, RASKOWSKY M, BLAKE RL, ET AL: TL-61 versus Rohadur orthoses in heel spur syndrome. JAPMA 81: 439, 1991. 12. MORAROS J, HODGE W: Orthotic survey. Preliminary results. JAPMA 83: 139, 1993. 13. SAGGINI R, GIAMBERARDINO MA, GATTESCHI L, ET AL: Myofascial pain syndrome of the peroneus longus: biomechanical approach. Clin J Pain 12: 30, 1996. 14. LYNCH DM, GOFORTH WP, MARTIN JE, ET AL: Conservative treatment of plantar fasciitis. A prospective study. JAPMA 88: 375, 1998. 15. SAXENA A, HADDAD J: The effect of foot orthoses on patellofemoral pain syndrome. JAPMA 93: 264, 2003. 16. SOBEL E, LEVITZ SJ, CASELLI MA: Orthoses in the treatment of rearfoot problems. JAPMA 89: 220, 1999. 17. WOODBURN J, BARKER S, HELLIWELL PS: A randomized controlled trial of foot orthoses in rheumatoid arthritis. J Rheumatol 29: 1377, 2002. 18. SLATTERY M, TINLEY P: The efficacy of functional foot orthoses in the control of pain in ankle joint disintegration in hemophilia. JAPMA 91: 240, 2001. 19. ROOT ML: How was the Root Functional Orthotic Developed? Perspectives in Podiatry. Podiatry Arts Lab Inc. Los Angeles, 1981. 20. ROOT ML, ORIEN WP, WEED JH: Normal and Abnormal Function of the Foot, Vol 2, Clinical Biomechanics Corporation, Los Angeles, 1977. 21. ROOT ML, ORIEN WP, WEED JH, ET AL: Biomechanical Examination of the Foot, Vol 1, Clinical Biomechanics Corporation, Los Angeles, 1971. 22. ANTHONY R: The Manufacture and Use of the Functional Foot Orthoses, Karger, Basel, 1990. 23. CURRAN M, PRATT D: Prescription Orthoses, in Clinical Skills in Treating the Foot, 2nd Ed, ed by WA Turner, LM Merriam, Churchill Livingstone, London, 2005. 24. HUNTER S, DOALN M, DAVIS J: Foot Orthotics in Therapy and Sport, 1st Ed, Human Kinetics, United Kingdom, 1995. 25. MICHAUD TC: Foot Orthoses and Other Forms of Conservative Foot Care, 2nd Ed, ed by TC Michaud, Lippincott Williams & Wilkins, Baltimore, 1997. 26. PHILPS JW: The Functional Foot Orthosis, 1st Ed, Elsevier Science, London, 1990. 27. RESSEQUE B. Orthotic Management, in Introduction to Podopaediatrics, 2nd Ed, ed by P Thomson, R Volpe, Churchill Livingstone, London, 2001. 28. VALMASSY RL: Lower Extremity Treatment Modalities for the Pediatric Patient in Clinical Biomechanics of the Lower Extremities, ed by RL Valmassy, Mosby, St. Louis, 1996. 29. VALMASSY R, SUBOTNICK SI: Orthoses, in Sports Medicine of the Lower Extremity, 2nd Ed, ed by SI Subotnick, Churchill Livingstone, London, 1999. 30. WEED JH, RATLIFF FD, ROSS SA: Biplanar grind for rearfoot posts on functional orthoses. JAPA 69: 35, 1979. 31. NORRIS CM: Biomechanics of the Lower Limb, in Sports Injuries, Diagnosis and Management for Physiotherapists, Butterworth Heinemann, Oxford, 1993. 32. MCPOIL TG, HUNT GC: Evaluation and management of foot and ankle disorders: present problems and future directions. J Orthop Sports Phys Ther 21: 381, 1995. 33. LANDORF K, KEENAN AM, RUSHWORTH R: Foot orthoses prescription habits of Australian and New Zealand podiatric physicians. JAPMA 91: 174, 2001. 34. ROOT ML, ORIEN WP, WEED JH: Neutral Position Casting Techniques, Vol 3, Clinical Biomechanics Corporation, Los Angeles, 1978. 35. PHILLIPS RD, CHRISTECK R, PHILLIPS RL: Clinical measurement of the axis of the subtalar joint. JAPMA 75: 119, 1985. 36. DANANBERG HJ: Functional hallux limitus and its relationship to gait efficiency. JAPMA 76: 648, 1986. 37. DANANBERG HJ: Gait style as an etiology to chronic postural pain: part 1. Functional hallux limitus. JAPMA 83: 433, 1993. 38. DANANBERG HJ: Gait style as an etiology to chronic postural pain: part 2. Postural compensatory processes. JAPMA 83: 615, 1993. 39. DANANBERG HJ: Lower Back Pain: A Repetitive Motion Injury, in Movement, Stability and Low Back Pain: The Role of the Sacroiliac Joint, ed by A Vleeming, V Mooney, CJ Snijders, et al, Churchill Livingstone, Edinburgh, 1997. 40. DANANBERG HJ: Lower Extremity Mechanics and Their Effect on Lumbosacral Function, in The Spine, State of the Art Reviews, Harley and Belfus Inc, Philadelphia, 1995. 41. DANANBERG H: Question and Answer Session. The Podiatric Biomechanics Group Focus 7: 7, 1999. 42. DANANBERG H: Sagittal Plane Biomechanics, in Sports Medicine of the Lower Extremity, 2nd Ed, ed by SI Subotnick, Churchill Livingstone, London, 1999. 43. DANANBERG H: Biomechanics Summer School 2000, Conference Notes. 44. KIRBY KA: Methods for determination of positional variations in the subtalar joint axis. JAPMA 77: 228, 1987. 45. KIRBY KA: Subtalar joint axis location and rotational equilibrium theory of foot function. JAPMA 91: 465, 2001. 46. KIRBY KA: Biomechanics of the normal and abnormal foot. JAPMA 90: 30, 2000. 47. FULLER EA: The windlass mechanism of the foot: a mechanical model to explain pathology. JAPMA 90: 35, 2000. 48. FULLER EA: Center of pressure and its theoretical relationship to foot pathology. JAPMA 89: 278, 1999. 49. FULLER E: Lecture Notes: The Tissue Stress Paradigm for Foot Biomechanics. The Podiatric Biomechanics 324 July/August 2009 Vol 99 No 4 Journal of the American Podiatric Medical Association

Group. November, 1999. 50. KIRBY KA: Foot and Lower Extremity Biomechanics: A Ten Year Collection of Precision Intricast News Letters, Precision Intricast Incorporated, Payson, Arizona, 1997. 51. KIRBY KA: The medial heel skive technique: improving pronation control in foot orthoses. JAPMA 82: 177, 1992. 52. KIRBY KA: Foot and Lower Extremity Biomechanics II: Precision Intricast Newsletters, 1997-2002, Precision Intricast, Incorporated, Payson, Arizona, 2002. 53. MCNERNEY JE: Football Injuries, in Sports Medicine of the Lower Extremity, 2nd Ed, ed by SI Subotnick, Churchill Livingstone, London, 1999. 54. PERRY J: Gait Analysis: Normal and Pathologic Function, Slack, Thorofare, NJ, 1992. 55. SARRAFIAN SK: Functional characteristics of the foot and plantar aponeurosis under tibio-fibular loading. Foot Ankle 8: 10, 1987. 56. AQUINO A, PAYNE C: The role of the reverse windlass mechanism in foot pathology. Aust J Pod Med 34: 32, 2000. 57. HICKS JH: The mechanics of the foot II. The plantar aponeurosis and the arch. J Anat 88: 25, 1954. 58. SARRAFIAN SK: Retaining Systems and Components, in Anatomy of the Foot and Ankle. Descriptive, Topographic, Functional, 2nd Ed, ed by SK Sarrafian, Lippincott, Philadelphia, 1993. 59. CHEUNG JT, AN KN, ZHANG M: Consequences of partial and total plantar fascia release: a finite element study. Foot Ankle Int 27: 125, 2006. 60. PANJABI M, WHITE A: Biomechanics in the Musculoskeletal System, 1st Ed, p 151, Churchill Livingstone, London, 2001. 61. BORRELLI AH: Planar dominance: a major determinant in flatfoot stabilization. Clin Podiatr Med Surg 16: 407, 1999. 62. CARTER N, HARRADINE PD, BEVAN LJ: Podiatric biomechanics: part 2. The role of proximal muscle balance. Br J Podiatr 11: 53, 2003. 63. TEITZ CC, HU SS, ARENDT EA: The female athlete: evaluation and treatment of sports-related problems. J Am Acad Orthop Surg 5: 87, 1997. 64. BEVAN LS, HARRADINE PD, DURRANT B: The effect of temporary immobilisation of the 1st metatarsophalangeal joint upon in-shoe gait analysis parameters: a preliminary study. Br J Podiatr 7: 54, 2004. 65. BOSJEN-MØLLER F: Calcaneocuboid joint stability of the longitudinal arch of the foot at high and low gear push off. J Anat 129: 165, 1979. 66. CLOUGH JG: Functional hallux limitus and lesser-metatarsal overload. JAPMA 95: 593, 2005. 67. CAMASTA CA: Hallux limitus and hallux rigidus. Clinical examination, radiographic findings, and natural history. Clin Podiatr Med Surg 13: 423, 1996. 68. COUGHLIN MJ, SHURNAS PS: Hallux rigidus: demographics, etiology, and radiographic assessment. Foot Ankle Int 24: 731, 2003. 69. ROUKIS TS, SCHERE PR, ANDERSON CF: Position of the first ray and motion of the first metatarsophalangeal joint. JAPMA 86: 538, 1996. 70. HARRADINE PD, BEVAN LS: The effect of rearfoot eversion upon maximal hallux dorsiflexion: a preliminary study. JAPMA 90: 390, 2000. 71. CORRIGAN B, MAITLAND G: Practical Orthopaedic Medicine, 1st Ed, p 113, Butterworth-Heinemann, Oxford, 1983. 72. WROBEL JS, CONNOLLY JE, BEACH ML: Associations between static and functional measures of joint function in the foot and ankle. JAPMA 94: 535, 2004. 73. MCKENZIE RA: The Lumbar Spine, Mechanical Diagnosis and Therapy, Spinal Publications Limited, New Zealand, 1981. 74. MAITLAND GD: Vertebral Manipulation, Butterworth-Heinemann, Oxford, 2000. Journal of the American Podiatric Medical Association Vol 99 No 4 July/August 2009 325